Wrong thc is not highly hallucinating is pyschotic, not the same thing. People don't hallucinate high on weed because of the thc
Hi yes, I have gel & drink tea & oral drops. I do believe it's helping! Esp oral drops. I've had few days waking without pain & quick pain relief on taking. Now I've noticed a bit more energy too. It's not cured me I still flare if overdo it. It's very expensive I take max strength £300 for 10ml but I only use 2 drops a day occasionally 5-10. Think will last me 3-4months this way.
It helped me! Something actually made the pain subside. It is morning and my back doesn't ache! I don't know how it worked, but it did. I'm bringing some to my massage therapist and will continue to use it.
Hi..since I last wrote I thoroughly researched CBD and became confused with all the distributors, information, strengths, claims, etc. However, I found a seller in the U.S. whom I trust because I have done business with her other company for many years. It's a long story and I'll be glad to tell you directly in an email, but I don't want to clog this thread with so much information.
Quite a story. I'm on the prednisone until the docs figure out what's wrong with my brain, with a PET scan. Who knows what I'll be put on next.
Cannabis was first described as a possible treatment for FMF in 1997. In a double-blind placebo-controlled crossover trial conducted on a single patient with FMF, THC was found to markedly reduce the need for opiate-based painkillers. However, it doesn’t appear that any further research has been conducted.
(South Africa, Cape Town)
Ehlers-Danlos syndrome (EDS) can cause hypermobile, unstable joints, hyperelastic skin, chronic pain, muscle spasms, deformities of the joint and spine, and cardiovascular complications. EDS is caused by mutations in certain key genes, which control the expression of proteins crucial to collagen production.
I have SOJGREN’S SYNDROME I’M TOLD.THERE IS NO.CURE. .SO , I THERE’S NO.CURE WHY pump me full full of medication 14 pills a day 3 chemo medications for antidepressants Marinol because it’s the only thing legal in Oklahoma at the time why make me into a cauldron of poison and soup if there’s no cure for it I don’t understand I don’t want to be a guinea pig
MDF is not generally thought to be fatal, but causes discomfort and difficulty with breathing and eating. There is no standard pharmaceutical treatment, but symptoms can be quickly relieved by temporarily blocking the phrenic nerve that runs past the lungs to reach the diaphragm.
In a 2013 study into neuropathic pain and hypersensitivity resulting from NMO, the endocannabinoids 2-AG and anandamide were both elevated in patients compared to healthy controls. The authors concluded that these endocannabinoids are released at higher levels in NMO sufferers, in order to reduce pain and sensitivity and prevent hyperalgesia.
In 2016, 6-year-old Harper Elle Howard tragically lost her battle against CDKL5. At just two weeks old, she began to experience life-threatening seizures that failed to respond to conventional treatments.
Biochanin A, a naturally occurring inhibitor of fatty acid amide hydrolase.
Research suggests that Cannabidiol may exert some of its pharmacological action through its inhibition of fatty acid amide hydrolase (FAAH), which may in turn increase the levels of endocannabinoids, such as anandamide, produced by the body. It has also been speculated that some of the metabolites of Cannabidiol/CBD have pharmacological effects that contribute to the biological activity of CBD.
Cannabidiol (INN: CBD) is one of at least 113 active cannabinoids identified in cannabis. It is a major phytocannabinoid, accounting for up to 40% of the plant’s extract.
Cannabidiol/CBD does not appear to have any intoxicating effects such as those caused by THC in marijuana, but may have effects on anxiety and an anti-psychotic effect.
Nicotine and biochanin A, but not cigarette smoke, induce anti-inflammatory effects on keratinocytes and endothelial cells in patients with Behçet’s disease. Moreover, biochanin A is likely to exhibit similar and even more profound results than nicotine.
Furthermore, (−)-CBD and (+)-CBD, as well as the natural metabolite 7-OH-CBD, inhibited fatty acid amide hydrolase with IC50 values of 27.5, 63.5, and 34.0 μM, respectively, but the 7-COOH-CBD metabolite was inactive (IC50 > 100 μM); also, anandamide uptake by rat basophilic leukemia cells was inhibited by (−)-CBD and by its 7-OH metabolite with IC50 values of 22.0 and ∼50 μM, respectively, as well as by (+)-CBD (IC50=17.0 μM) but 7-COOH-CBD was inactive (IC50 > 50 μm).